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Groups evidently differed in their neurological status (mJOA DCM 10.1 ± 3.3, TAAA 17.3 ± 1.2, p less then .001; NDI DCM 47.4 ± 19.7, TAAA 5.3 ± 8.6, p less then .001). There were no particular differences in age and gender distribution. However, we detected statistically significant differences in concentrations of mediators between the groups Angiopoietin 2 (DCM 267.1.4 ± 81.9, TAAA 408.6 ± 177.1, p less then .001) and VEGF C (DCM 152.2 ± 96.1, TAAA 222.4 ± 140.3, p = .04). DCM patients presented a mild to moderate BSCB disruption,
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