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To investigate this topic, we employed CRISPR/Cas9 to produce cyp11c1 (11β-hydroxylase) mutant zebrafish lines. Our research confirms recently posted conclusions from a different cyp11c1-/- mutant zebrafish range, also reports book components of the phenotype brought on by lack of Cyp11c1 function. We report that Cyp11c1-deficient zebrafish display predominantly female additional intercourse qualities, but may have either ovaries or testes. Furthermore, we observe
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