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Appropriately, focusing on HuR by its inhibitor DHTS inhibited splenic Th17 cellular differentiation and paid off experimental autoimmune encephalomyelitis seriousness. In sum, we revealed the molecular process of HuR controlling Th17 cell functions, underscoring the healing worth of HuR for treatment of autoimmune neuroinflammation. Copyright © 2020 because of the United states Association of Immunologists, Inc.Inflammatory bowel diseases tend to be associated with complex changes in microbiota composi