https://www.selleckchem.com/products/go-203.html
The AVI-dependent reduction in CAZ MIC was well characterised in both bacterial isolates (r ≥ 0.98). In the HFIM, sustained suppression of KP118 (T MICi = 100%) was observed over 5 days, but not with KP286 (T MICi 100%). These observations are consistent with the clinical course of the patients. The discordant patient outcomes could be potentially explained by MIC profiling of CAZ/AVI. This method appears to be more robust than conventional susceptibility testing in predicting positive clinical outcome of CAZ/AVI therapy,
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