https://www.selleckchem.com/products/harmine.html
t sunitinib could upregulate the expression of STAT1 by inhibiting the phosphorylation site Tyr751 of PDGFR, thereby specifically inducing the apoptosis of the primary heterotopic mesenchymal endometrium. The in vitro experiments revealed that sunitinib could upregulate the expression of STAT1 by inhibiting the phosphorylation site Tyr751 of PDGFR, thereby specifically inducing the apoptosis of the primary heterotopic mesenchymal endometrium. Pre-eclampsia (PE), preterm birth (PT and intra-uterine growth restriction (IUGR) affect 5%-1
Like
Comment
Share
