https://www.selleckchem.com/products/azd2014.html
41% (21/184) in patients with mutations. Glycosaminoglycan-related pathways were significantly upregulated in the mutant group. -mutated patients had lower TMB and PD-L1 protein levels than those in wild-type patients. Increase immature DCs infiltration and decreased NK CD56dim, T gamma delta, cytotoxic, and Th2 cell infiltration were the main immune changes in -mutated patients. Patients with -MAPK co-mutations had higher levels of TMB and PD-L1 protein expression. Meanwhile, the co-mutated patients had a similar immune microenvironmen
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