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In this research we investigated the effects of β-sitosterol on influenza virus-induced irritation and acute lung injury additionally the molecular components. We display that β-sitosterol (150-450 μg/mL) dose-dependently suppresses inflammatory response through NF-κB and p38 mitogen-activated necessary protein kinase (MAPK) signaling in influenza A virus (IAV)-infected cells, which was accompanied by diminished induction of interferons (IFNs) (including Type we and III IFN). Al
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