https://www.selleckchem.com/products/apg-2449.html
Traumatic brain injury (TBI) patients are reported to suffer from long-term sensorimotor dysfunction, with gait deficits evident up to two years after the initial brain trauma. Experimental TBI including rodent models of penetrating ballistic-like brain injury and severe controlled cortical injury (CCI) can induce impairments in static and dynamic gait parameters. It is reported that the majority of deficits in gait-related parameters occur during the acute phase post-injury, as functional outcomes return toward baseline levels at chro
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