https://cl-amidinechemical.com..../the-effect-associat
The development of miR-216a inhibited MPP + -induced neuronal apoptosis. The key manifestations were the reduced quantities of positive price of Tunel cells, caspase 3 activity and nucleosome enrichment element. Bax was a direct target of miR-216a. In addition, Bax overexpression reversed the consequences of miR-216a on neural cells. Bax downstream elements were also involved in miR-216a regulation of MPP + -triggered neuronal apoptosis. miR-216a regulated th
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