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As key motorists of neuroinflammation, we hypothesized that microglia directly regulate acidosis after TBI, and thus, intensify neurologic outcomes. Making use of a controlled cortical effect model in adult male mice we demonstrate that intracellular pH in microglia and extracellular pH surrounding the lesion web site are notably paid down for months after damage. Microglia proliferation and creation of reactive oxygen species (ROS) were also increased throughout the first week, mirroring th
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