https://www.selleckchem.com/pr....oducts/dansylcadaver
ROS activated p38MAPK to enter the nucleus, which was attenuated by PPAR . In OA chondrocytes, IL-1 induced COX-2 and PGE expression via activation of NOX2, which led to ROS production and MAPK activation. The activation of PPAR exerted protective roles in the pathogenesis of OA. In OA chondrocytes, IL-1β induced COX-2 and PGE2 expression via activation of NOX2, which led to ROS production and MAPK activation. The activation of PPARγ exerted protective roles in the pathogenesis of OA.Mechanical thrombectom
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