https://jakenzyme.com/excessiv....e-electrophysiologic
The present study aimed to elucidate the involvement of SAA in BBB impairment in an in vitro BBB design utilizing rat brain microvascular endothelial cells (RBECs). We demonstrated that Apo-SAA notably reduced transendothelial electric opposition (TEER) and enhanced sodium fluorescein (Na-F) permeability in RBEC monolayers. Apo-SAA also decreased claudin-5 expression levels in RBECs. Furthermore, the Apo-SAA-mediated impairment for the BBB with reduced claudin-5 expression wa
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