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We investigated the results of CGA on endothelial cell dysfunction in diabetic mice in addition to mechanistic role of nuclear factor erythroid-related element 2 (Nrf2) in the antioxidant aftereffect of CGA. Diabetic (db/db) mice were provided regular chow or chow containing 0.02% CGA for 12 weeks. Peoples umbilical vein endothelial cells (HUVECs) and mouse aortas had been addressed with regular or large glucose. CGA treatment induced upregulation of Nrf2 in HUVECs i
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