https://www.selleckchem.com/products/pt2399.html
The plasma level of HMOX1 in patients with CHF was significantly higher compared with the control group and was highly correlated with cardiac function in CHF patients. CDR1as was shown to act as a sponge for miR-135a and miR-135b and regulated the proliferation and apoptosis of human cardiomyocytes through the miR-135a/HMOX1 and miR-135b/HMOX1 signaling axes. Conclusion CDR1as is a potential biomarker of CHF that is mechanistically involved in the disease pathogenesis and participates in regulating the occurrence and development of CHF
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