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Aside from concentrating on the major inhibitory effectation of glial scar associated proteoglycans like chondroitin sulfate plus the reactive glial cell secreted inflammatory and growth inhibitory molecules, the other significant regeneration stalling component that arises after severe CNS injury is myelin as well as its associated proteinaceous debris comprising myelin-associated glycoprotein (MAG), neurite outgrowth inhibitor protein (NOGO), and oligodendrocyte myelin glycoprotein