https://www.selleckchem.com/mTOR.html
Finally, the review ends up with subjective views on present limitations and some appropriate advice for future research directions. Neurological injury following successful resuscitation from sudden cardiac arrest (CA) is common. The pathophysiological basis of this injury remains poorly understood, and treatment options are limited. Microglial activation and neuroinflammation are established contributors to many neuropathologies, such as Alzheimer disease and traumatic brain injury, but their potential role in post-CA injury has only recently bee
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