https://bche-receptor.com/inde....x.php/breaks-in-the-
Knockdown of LINC00266-1 suppressed the proliferative and metastatic abilities, and promoted the apoptosis in OS cells. Besides, knockdown of LINC00266-1 dramatically alleviated the development of OS in vivo. MiR-548c-3p had been the sponge miRNA of LINC00266-1, that was in a position to reverse the regulating ramifications of LINC00266-1 on OS mobile phenotypes. Moreover, miR-548c-3p bound into the 3'-UTR of SMAD2 and thus downregulated SMAD2. Overexpression of SMAD2 partly reversed the r
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