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To explore the effect and potential mechanism of glycyrrhizin (GL) by inhibiting high mobility group box 1 (HMGB1) on glial scar formation after spinal cord injury (SCI) in rats. Seventy-two female Sprague Dawley rats were randomly divided into sham group ( =12), SCI model group (SCI group, =36), GL intervention group (SCI+GL group, =12), and nuclear factor κB (NF-κ inhibitor [pynolidine dithiocarbamate (PDTC)] intervention group (SCI+PDTC group, =12). The SCI models of SCI group, SCI+GL group, and SCI+PDTC group were made by modified
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