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Compounds targeting tumor cell metabolism could become potential treatment options for glioblastoma multiforme. The mechanisms underlying the contribution of the heparan sulfate proteoglycan syndecan-1 to liver tissue injury and to crucial biological processes, such as fibrogenesis, remain to be elucidated. Therefore, we investigated the immunohistochemical expression of syndecan-1 in chronic liver diseases (CLDs) and its probable role in hepatic fibrosis. Immunohistochemistry was performed on formalin-fixed, paraffin-embedded tissue se
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