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Flow cytometry and lipid quantitation results revealed that Ox-LDL caused lipid and cholesterol accumulation in HUVECs. Western blot outcomes indicated that Ox-LDL increased the phrase of autophagy-related proteins P62 and LC3-II in a concentration-dependent manner. The cathepsin D task assay revealed that Ox-LDL inhibited the big event of cathepsin D. HNG pretreatment reduced lipid and cholesterol aggregation in HUVECs caused by Ox-LDL, enhanced LC3-II necessary protein level, decreased P62 ne
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