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K channels are of great interest to epilepsy research as mutations in their genes are found in humans with inherited epilepsy. At the level of cellular physiology, K+channels control neuronal intrinsic excitability and are the main contributors to membrane repolarization of active neurons. Recently, a genetically modified voltage-dependent K channel has been patented as a remedy for epileptic seizures. We review the role of potassium channels in excitability, clinical and experimental evidence for the association of potassium channelopat
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