https://s38093antagonist.com/o....utcomes-of-a-recentl
We found that GPX8 appearance had been induced because of the epithelial-mesenchymal transition (EMT) program . More over, in cancer of the breast patients, GPX8 expression notably correlated with understood mesenchymal markers and poor prognosis. Strikingly, GPX8 knockout in mesenchymal-like cells (MDA-MB-231) resulted in an epithelial-like morphology, down-regulation of EMT traits, and loss in cancer tumors stemness features. In inclusion, GPX8 knockout dramatically delayed tumor initiation and decreased its
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