https://www.selleckchem.com/products/unc8153.html
Our study highlights a role for α-synuclein in disturbing proteostasis which may contribute to neurodegeneration in vivo.BACKGROUND Synovitis is implicated in the severity and progression of pain and structural pathology of osteoarthritis (OA). Increases in inflammatory or immune cell subpopulations including macrophages and lymphocytes have been reported in OA synovium, but how the particular subpopulations influence symptomatic or structural OA disease progression is unclear. Two therapies, hyaluronan (HA) and mesenchymal stem cells (
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