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This research examined whether thioridazine changed Ca2+ signaling and viability in HepG2 man hepatocellular carcinoma cells. Ca2+ concentrations in suspended cells were assessed using the fluorescent Ca2+-sensitive dye fura-2. Cell viability had been analyzed by WST-1 assay. Thioridazine at concentrations of 25-100 μM induced [Ca2+]i rises. Ca2+ treatment paid down the signal by 20%. Thioridazine (100 μM) induced Mn2+ influx suggesting of Ca2+ entry. Thioridazine-induced Ca2+ entry w
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