https://www.selleckchem.com/pr....oducts/compstatin.ht
ing of skeletal muscle regeneration through the identification of Fbxl2 as both a critical regulator of myogenic proliferative processes and a susceptible gene target during inflammatory stimulation by TNFα in skeletal muscle. Modulation of Fbxl2 activity may have relevance to disorders of muscle wasting associated with sustained pro-inflammatory signaling. Copyright © 2020 American Society for Microbiology.Defects in the spindle assembly checkpoint (SAC) can lead to aneuploidy and cancer. Sphingolipids have important roles in many c
Like
Comment
Share
