https://www.selleckchem.com/pr....oducts/GSK461364.htm
Employing in silico analyses, we ranked polymorphisms in C57Bl/6 N substrain and selected genes Crb1, Cyfip2, Adamts12, Plk1 and Herpud2 as the most probable candidates, whose product dysfunction might be responsible for the ectopic distribution of CSF-cNs. Furthermore, segregation analysis of F2 progeny of parental C57Bl/6 N and Balb/C mice revealed that polymorphic loci of Crb1and Cyfip2 underlie the ectopic position of CSF-cNs in the spinal cord of C57Bl/6 N mice. This article is protected by copyright. All rights reserved. © 2020
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