https://www.selleckchem.com/MEK.html
Alternative polyadenylation (APA) is a pervasive posttranscriptional mechanism regulating gene expression. However, the specific dysregulation of APA events and its potential biological or clinical significance in lung adenocarcinoma (LUAD) remain unclear. Here, we collected RNA-Seq data from two independent datasets GSE40419 ( = 146) and The Cancer Genome Atlas (TCGA) LUAD ( = 542). The DaPars algorithm was employed to characterize the APA profiles in tumor and normal samples. Spearman correlation was used to assess the effects of APA regulators on
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