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CDK4 was identified as a potential target for miR-486-5p. LncRNA LINC01194 was able to inhibit miR-486-5p expression and upregulate the expression level of CDK4. Finally, the results of in vivo animal models confirmed that lncRNA LINC01194 promoted NSCLC progression by modulating the miR-486-5p/CDK4 axis. Conclusion LncRNA LINC01194 promoted the progression of NSCLC by modulating the miR-486-5p/CDK4 axis. © 2020 Xing et al.Background In a data mining search for potential therapeutic targets to improve the outcome of rectal cancer, we identified PCSK1 as the
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