https://www.selleckchem.com/MEK.html
BACKGROUND Chronic inflammation associated with delayed diabetic wound healing is induced by disturbed polarization of macrophages derived mainly from predisposed progenitor cells in bone marrow. Docosahexaenoic acid plays a critical role in regulating the function of macrophage progenitor cells. The authors evaluated whether docosahexaenoic acid accelerates diabetic wound healing in rats. METHODS Streptozotocin-induced diabetic rats divided into control and docosahexaenoic acid-treated groups (n = 1 were subjected to paired dorsal skin wounds. Do
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