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In inclusion, T-17 promoted the cancer cell autophagy as evidenced with increased expression of Beclin-1 and decreased p62 in western blot and formation of GFP-LC3 puncta. Furthermore, T-17-induced autophagy reduced gastric cancer mobile apoptosis as examined by pharmacological autophagy inhibitors and ATG5 siRNA usage. Importantly, the activation of JNK pathway ended up being simultaneously involved in T-17-induced apoptosis and autophagy. Taken collectively, th

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