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Numerous experimental and clinical studies have shown that oxidative stress (OS) plays an important role in the development of epilepsy. The epileptic seizures themselves are caused by OS and, under conditions of a hypermetabolic state, cause an intensive production of reactive oxygen species (ROS) in the brain. The reason for this is called a change in the energy balance in the brain. According to the literature, ROS and other markers of oxidative stress were observed in the brain after seizures. Based on the above data, the aim of our
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