https://www.selleckchem.com/pr....oducts/disodium-Crom
Transcriptome pathway analysis highlighted mitochondrial dysfunction and altered fatty acid oxidation as key molecular perturbations associated with zileuton exposure, and suggested that interindividual differences in cytochrome P450 metabolism, glutathione-mediated detoxification, and FXR signaling may contribute to zileuton-induced liver injury. Taken together, DO mice provided a platform for investigating mechanisms of toxicity and resistance in context of zileuton-induced liver injury which may lead to targeted therape
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