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Focal chromosomal aberrations included amplifications of 5p15.33 (ZDHHC11 and 7p14.1 (TARP) as well as deletion of 9p21.3 (CDKN2A/. The protein expression levels of ZDHHC11B and TARP in EACSCC tissues were validated by immunohistochemistry. Moreover, WES of the primary and relapsed tumors from a case of synchronous bilateral EACSCC showed the intrapatient genetic heterogeneity of EACSCC. In summary, this study provides the first evidence for genetic alterations of EACSCC. Our findings suggest that EACSCC mostly resembles other SCCs.
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