https://www.selleckchem.com/pr....oducts/mpi-0479605.h
The aim of this study was to investigate the effects of trichostatin A (TSA) on cervical cancer and the related mechanisms. The HeLa and Caski cervical cancer cell lines were treated with different concentrations of TSA. Cell viability was measured by MTT assays. Cell apoptosis was analysed using flow cytometry. Expression of transient receptor potential cation channel, subfamily V, member 6 (TRPV6), protein arginine methyltransferase 5 (PRMT5), and stanniocalcin 1 (STC1) was determined by qRT-PCR and Western blotting. Protein level
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