https://www.selleckchem.com/products/arv-825.html
BACKGROUND Chronic inflammation associated with delayed diabetic wound healing is induced by disturbed polarization of macrophages derived mainly from predisposed progenitor cells in bone marrow. Docosahexaenoic acid plays a critical role in regulating the function of macrophage progenitor cells. The authors evaluated whether docosahexaenoic acid accelerates diabetic wound healing in rats. METHODS Streptozotocin-induced diabetic rats divided into control and docosahexaenoic acid-treated groups (n = 1 were subjected to paired dorsal sk
Like
Comment
Share
