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Curve-shift experiments were consistent with competition between SV-III-130 and DA. Two mutations in the additional binding pocket (V91A and E95A) of D2R decreased antagonistic effectiveness and increased recovery from SV-III-130 antagonism, whereas a third mutation (L94A) only increased recovery. Our outcomes claim that the secondary binding pocket influences data recovery from inhibition because of the studied aripiprazole analogues. We propose a mechanism, supported by in
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