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8 ± 7.6 mm). There was no significant change in peak contact stress (p = 0.901) in either the retroverted (0°) or anteverted (30°) conditions relative to normal version (15°) under identical gait-related loading conditions. While abnormalities in patient gait and resultant joint loading caused by femoral version abnormalities may contribute to hip pain, the present findings would suggest that future joint degeneration in hips with version abnormalities are not simply the result of abnormal contact stress induced by joint incongruity due t
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