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Endothelial dysfunction is an important part of preeclampsia like premature ovarian insufficiency (POI), which is stated that placental growth element (P1GF) and dissolvable fms-like tyrosine kinase receptor-1 (sFlt-1) amounts are very important in preeclampsia. Extra-placental resources for P1GF and sFlt-1 are also identified, including different mobile kinds. In animal models of POI, niacin treatment inhibited follicular apoptosis under hazardous problems while somewhat reducing cu

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