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CONCLUSION Collectively, our findings demonstrate that atypical E2Fs can override cell-cycle entry and progression governed by other E2F-family members, and suggest that this property can be exploited to inhibit proliferation of neoplastic hepatocytes when growth and development have subsided during adulthood. This article is protected by copyright. All rights reserved.The increasing complexity of health care systems creates a growing urgency to collaborate across disciplines. Insight into the underlying processes or, as described in th
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