https://www.selleckchem.com/products/gdc-0084.html
pogenesis and HPS/FGL1-mediated cell survival in -mutated pancreatic cancer cells. Results of this study indicate that HPS is highly expressed by KRAS-mutated PDAC cells, and HPS/FGL1 plays a crucial role in altering lipid metabolism and increasing cell growth in pancreatic cancer. EPA supplements could potentially inhibit or reduce ACC-1-involved lipogenesis and HPS/FGL1-mediated cell survival in KRAS-mutated pancreatic cancer cells.Human septins comprise a family of 13 genes that encode conserved GTP-binding proteins. They form nonpo
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