https://www.selleckchem.com/Bcl-2.html
We suggest that this is caused by a hyperactive amygdala → thalamic reticular nucleus circuit increasing activity in the PIm → MT via response gain modulation of the pulvinar and hence altering synaptic competition in area MT. We explore the probable timing of transfer in dominance of human MT from pulvinar to LGN/V1 driving circuitry and discuss the implications of the main hypothesis.Parkinson's disease (PD) is the second most common neurological disease having no specific medical test for its diagnosis. In this study, we consider PD detection b
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