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Appearing studies have uncovered that the interactions between MEG3 and proteins may take place in illness development. Galectin-1 affects cell motility, sign transduction and vascularization, and it is overexpressed in endometriosis. Our study could be the first to explore the part of MEG3-210 transcript in endometriosis also to reveal the regulating system mediated because of the interaction between MEG3-210 and Galectin-1. Materials and pract

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