https://www.selleckchem.com/products/vt104.html
Here, we summarize the current knowledge highlighting pathogenic mechanisms of the different disorders that compose acromelic dysplasias and provide an overview of the emerging biological roles of the individual proteins that are compromised. Finally, we develop a conceptual model of how these proteins may interact and form an "acromelic dysplasia complex" on fibrillin microfibrils in connective tissues of the musculoskeletal system.Clonorchis sinensis, an important fish-borne zoonotic trematode, is widely distributed in South-East Asia,
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