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Overexpression of vimentin notably suppressed the replication of on for the FMDV 3A have the effect of the discussion between 3A and vimentin while the 3A-vimentin interacting with each other is critical for the viral replication since full-length cDNA clone harboring mutations in 3A, that have been disrupt 3A-vimenin reactivity, could perhaps not create viable virus progeny. This study provides information that not only provides us an improved understanding of the process of FMDV replication
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