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The present research had been aimed to show the function of extracellular RNAs (exRNAs) in retinal ischemia reperfusion (I/R) damage, and evaluate whether RNase administration can effectivelyreduce I/Rinjury. A retinal I/R injury C57BL/6J wild-type mice model had been founded by elevating intraocular pressure for 1 h. All mice obtained 3 amounts of RNase or even the exact same dose of regular saline at different time things. After 7 days of reperfusion, retinal damage was quantified by countin
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