https://www.selleckchem.com/
asures in this cohort would clarify the robustness of these findings. Mesenchymal stem cells-conditioned medium (MSC-CM) provides a promising cell-free therapy for Alzheimer's disease (AD) mainly due to the paracrine of MSCs, but the precise mechanisms remain unclear. Studies suggests that mitochondrial dysfunction precedes the accumulation of amyloid-β plaques and neurofibrillary tangles, and involves in the onset and development of AD. In the present study, we evaluated the protective effects and explored the related-mitochondrial mechanisms of human umbi
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