https://www.selleckchem.com/pr....oducts/phorbol-12-my
Liver fibrosis risk is a heritable trait, the outcome of which is the net deposition of extracellular matrix by hepatic stellate cell-derived myofibroblasts. Whereas nucleotide sequence variations have been extensively studied in liver fibrosis, the role of copy number variations (CNV) in which genes exist in abnormal numbers of copies (mostly due to duplication or deletion) has had limited exploration. The impact of the XPO4 CNV on histological liver damage was examined in a cohort comprised 646 Caucasian patien
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