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Extra anti inflammatory therapeutic strategies tend to be consequently required. The co-stimulatory molecule CD40 and its ligand CD40L (CD154) have actually a central part in the legislation for the inflammatory reaction throughout the growth of atherosclerosis by modulating the discussion between resistant cells and between immune cells and non-immune cells. In this analysis, we discuss the role of the CD40-CD40L dyad in atherosclerosis, so we discuss current scientific
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