https://www.selleckchem.com/products/jq1.html
However, the fractions failed in upregulating either caspase-3 activation or cytochrome c release in the cancer cells. Overall, these results suggest that the cytotoxic effect of MC on the selected cancer cells is mediated by loss of mitochondrial function via loss of respiration leading to cell death rather than by the classical release of cytochrome c or caspase-3 activated apoptosis. Overall, these results suggest that the cytotoxic effect of MC on the selected cancer cells is mediated by loss of mitochondrial function via loss of respir
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