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Heart transplantation remains the definitive therapy of end-stage heart failure. Ischemia-reperfusion injury occurring during transplantation is a primary determinant of long-lasting results of heart transplantation and primary graft insufficiency. Modification regarding the nitric oxide/soluble guanylate cyclase/cyclic guanosine monophosphate signaling path appears to be the most promising on the list of pharmacological interventional options. We geared towards characterizing
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